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You might be curious as to why this disorder of repetition is called conduction aphasia.
Conduction aphasia usually is the result of damage to the left brain hemisphere, such as by stroke.
Conduction aphasia is a relatively mild language impairment, and most patients return to day-to-day life.
However, chronic conduction aphasia is possible, without transformation to other aphasias.
These tests are capable of identifying conduction aphasia with relative accuracy.
However, conduction aphasia remains a controversial topic from the understanding of its neurologic foundations.
Symptoms of conduction aphasia, as with other aphasias, can be transient, lasting only several hours or a few days.
Conduction aphasia can also be seen in cases of cortical damage without subcortical extensions.
Patients with conduction aphasia are typically able to speak fluently, but they do not have the ability to repeat what they hear.
Traumatic brain injury and tumors can also lead to localized lesions, with potential to cause conduction aphasia.
Individuals with Conduction aphasia have deficits in the connections between the speech-comprehension and speech-production areas.
There are all kinds of reasons why this explanation will not work; we mention it only to explain the origin of the term 'conduction aphasia'.
In cases of conduction aphasia, comprehension of spoken words and simple spoken sentences can be intact.
Conduction aphasia is another speech disorder that is similar to, but not the same as, apraxia of speech.
The brain damage causing conduction aphasia is often from a stroke, which can produce both localized and widespread damage.
Conduction aphasia is caused by damage in the language-dominant hemisphere (the left hemisphere in most individuals).
There have been no known autopsy cases in which conduction aphasia was shown to be the result of a focused arcuate fasciculus lesion.
The Wernicke-Lichtheim-Geschwind disconnection hypothesis thus became the prevailing explanation for conduction aphasia.
Conduction aphasia, also called associative aphasia, is a relatively rare form of aphasia.
Conduction aphasia: Characterized by poor speech repetition, this disorder is rather uncommon and happens when branches of the arcuate fasciculus are damaged.
Although patients who suffer from conduction aphasia have full comprehension of speech, as do AOS sufferers, there are differences between the two disorders.
Although the disconnection hypothesis explains many of the conditions associated with conduction aphasia, clinical evidence is lacking, and the Wernicke-Geschwind model has since become obsolete.
Recent research has challenged this notion on the basis that patients with conduction aphasia more often have lesions in the supramarginal gyrus or deep parietal matter.
Lesions in the language centers (and connections between) have been identified as potential inducers for conduction aphasia (and other language disorders, in general).
However, conduction aphasia is a mild aphasia, and conduction aphasics score highly on the WAB at baseline.