This may be because of the different types and doses of cholecystokinin used.

Ceruletide has very much in common with regards to action and composition to cholecystokinin.

Together with cholecystokinin, it is the second major source of negative feedback signals that stop eating behaviour.

Ceruletide is similar in action and composition to cholecystokinin.

Researchers have also found low cholecystokinin levels in bulimics.

The cholecystokinin B receptor responds to a number of ligands.

Blood was obtained by direct cardiac puncture for cholecystokinin assay.

So how do you get more cholecystokinin?

Cholecystokinin can have indirect effects on sleep regulation.

Development of cholecystokinin binding sites in rat upper gastrointestinal tract.

You've probably never heard of cholecystokinin, but it's one of your best weight-loss pals.

Potent and sustained satiety actions of a cholecystokinin octapeptide analogue.

Autoradiographic and functional development of gastric cholecystokinin receptors in the rat.

In vitro response of rat gastrointestinal segments to cholecystokinin and bombesin.

They also cause the release of cholecystokinin, which stimulates the digestion of fat and protein.

This causes RB-101 to be strongly synergistic with cholecystokinin antagonists.

There are several different types which secrete the hormones secretin, cholecystokinin and several others.

Gastric and nongastric mechanisms for satiety action of cholecystokinin.

Two brain cholecystokinin receptors: implications for behavioral actions.

It is required for interaction of the cholecystokinin A receptor with its corresponding hormonal ligand.

The first hormone to be studied by Ondetti's group was cholecystokinin, a digestion hormone.

Cholecystokinin has been shown to interact with orexin neurons which control appetite and wakefulness (sleep).

The gallbladder releases the bile in response to a hormone called cholecystokinin, which is released from the small intestine.

This hormone is cholecystokinin (kol'eh-sis-toh-ky'nin; "to move the gallbladder" G).

Gastric loads potentiate inhibition of food intake produced by a cholecystokinin analogue.

This may be because of the different types and doses of cholecystokinin used.

Together with cholecystokinin, it is the second major source of negative feedback signals that stop eating behaviour.

Ceruletide has very much in common with regards to action and composition to cholecystokinin.

Researchers have also found low cholecystokinin levels in bulimics.

Ceruletide is similar in action and composition to cholecystokinin.

The cholecystokinin B receptor responds to a number of ligands.

Blood was obtained by direct cardiac puncture for cholecystokinin assay.

So how do you get more cholecystokinin?

Cholecystokinin can have indirect effects on sleep regulation.

Development of cholecystokinin binding sites in rat upper gastrointestinal tract.

You've probably never heard of cholecystokinin, but it's one of your best weight-loss pals.

They also cause the release of cholecystokinin, which stimulates the digestion of fat and protein.

This causes RB-101 to be strongly synergistic with cholecystokinin antagonists.

Potent and sustained satiety actions of a cholecystokinin octapeptide analogue.

Autoradiographic and functional development of gastric cholecystokinin receptors in the rat.

In vitro response of rat gastrointestinal segments to cholecystokinin and bombesin.

There are several different types which secrete the hormones secretin, cholecystokinin and several others.

It is required for interaction of the cholecystokinin A receptor with its corresponding hormonal ligand.

Gastric and nongastric mechanisms for satiety action of cholecystokinin.

Two brain cholecystokinin receptors: implications for behavioral actions.

The first hormone to be studied by Ondetti's group was cholecystokinin, a digestion hormone.

Cholecystokinin has been shown to interact with orexin neurons which control appetite and wakefulness (sleep).

The gallbladder releases the bile in response to a hormone called cholecystokinin, which is released from the small intestine.

This hormone is cholecystokinin (kol'eh-sis-toh-ky'nin; "to move the gallbladder" G).

Gastric loads potentiate inhibition of food intake produced by a cholecystokinin analogue.