The cell-cell adhesion molecule E-cadherin has been shown to be required for the radial intercalation of the deep cells.

This gene is a member of the cadherin superfamily whose members encode calcium-dependent cell-cell adhesion molecules.

Reduction of the activity of these cell-cell adhesion molecules allows for cells undergoing convergent extension to move more freely.

Toxins are produced by S. aureus and target desmoglein, which is a desmosomal cell-cell adhesion molecule that is found in the upper levels of the epidermis.

Metastatic cells must mimic normal cell-cell interactions, through cell-cell adhesion molecules (CAMs) and integrins.

In addition to the non-canonical Wnt and PCP pathways involvement in convergent extension, the down-regulation of certain cell-cell adhesion molecules, such as C-cadherin and fibronectin/integrin interactions, may also play a role.

The cell-cell adhesion molecule E-cadherin has been shown to be required for the radial intercalation of the deep cells.

This gene is a member of the cadherin superfamily whose members encode calcium-dependent cell-cell adhesion molecules.

Reduction of the activity of these cell-cell adhesion molecules allows for cells undergoing convergent extension to move more freely.

Toxins are produced by S. aureus and target desmoglein, which is a desmosomal cell-cell adhesion molecule that is found in the upper levels of the epidermis.

Metastatic cells must mimic normal cell-cell interactions, through cell-cell adhesion molecules (CAMs) and integrins.

In addition to the non-canonical Wnt and PCP pathways involvement in convergent extension, the down-regulation of certain cell-cell adhesion molecules, such as C-cadherin and fibronectin/integrin interactions, may also play a role.