carbon monoxide haemoglobin
carbon monoxide hemoglobin
carboxyhaemoglobin
carboxylated haemoglobin
carboxylated hemoglobin

The half-life of nicotine and carboxyhaemoglobin is thought to be approximately two hours.

After smoking a single cigarette there is a significant nicotine and carboxyhaemoglobin boost which reaches its peak within 10 minutes.

Their skin was coloured cherry-pink from carboxyhaemoglobin, the stable compound formed in the blood by reaction with the gas.

It binds to haemoglobin much more readily than oxygen, leading to the formation of carboxyhaemoglobin: the blood can therefore carry less oxygen.

This is known as "Fetal carboxyhaemoglobin" (%FCOHb).

We observed limitations of SpO in 6 patients treated after inhalation of carbon monoxide (CO), whose carboxyhaemoglobin (COHb) on admission ranged between 29% and 41%.

The breath carbon monoxide level has been shown to have a close relationship with the level of CO in the blood known as carboxyhaemoglobin (%COHb) or "blood CO".

Carboxyhemoglobin (British English: Carboxyhaemoglobin) (COHb) is a stable complex of carbon monoxide and hemoglobin that forms in red blood cells when carbon monoxide is inhaled or produced in normal metabolism.

The following guideline values (ppm values rounded) and periods of time-weighted average exposures have been determined in such a way that the carboxyhaemoglobin (COHb) level of 2.5% is not exceeded, even when a normal subject engages in light or moderate exercise:

Depending on the nature of the myeloproliferative disorder, diagnostic tests may include red cell mass determination (for polycythemia), bone marrow aspirate and trephine biopsy, arterial oxygen saturation and carboxyhaemoglobin level, neutrophil alkaline phosphatase level, vitamin B (or B binding capacity) and serum urate.

Carboxyhaemoglobin concentrations have not been studied in direct relation to gastric secretion in man but this agent may cause relative anoxia of the cell; both anoxia and the thiocyanate ion (another cigarette product) have been shown to prevent the transformation by histamine of a resting parietal cell to its active acid secreting state.