bulbar palsy

In 1859, Wachsmuth changed the name to progressive bulbar palsy.

Bulbar palsy, a similar syndrome caused by the damage of lower motor neurons.

Bulbar Palsy is an assortment of signs and symptoms, not the name of a precise disease.

Progressive bulbar palsy is slow in onset, with symptoms starting in most patients around 50-70 years of age.

Progressive bulbar palsy (also known simply as PBP) is a medical condition.

Progressive bulbar palsy symptoms can include progressive difficulty with chewing, talking, and swallowing.

Deep tendon reflexes are usually hypoactive or absent, and ophthalmoplegia and bulbar palsy can occur.

Fazio-Londe disease, also called progressive bulbar palsy of childhood, is an inherited motor neuron disease found in children and young adults.

It produces rapidly progressive weakness of tongue, face and pharyngeal muscles in a clinical pattern similar to myasthenia bulbar palsy.

Progressive bulbar palsy of childhood (Fazio-Londe)

Progressive bulbar palsy is characterized by progressive paralysis of muscles innervated by cranial nerves.

It's part of a group of motor neuron diseases that includes progressive muscular atrophy, primary lateral sclerosis and progressive bulbar palsy.

The term Infantile progressive bulbar palsy is used to describe progressive bulbar palsy in children.

Infantile Progressive Bulbar palsy is a rare type of progressive bulbar palsy that occurs in children.

Mr. Bailey stepped down as chief state's attorney after he was told by doctors that he had progressive bulbar palsy, a rare disorder in which talking and swallowing become increasingly difficult.

Dr. Benjamin Brooks of Carolinas Medical Center said Basnight had progressive bulbar palsy and that, while someone could die from the condition, it was also possible to live a long time.

Clinicians frequently think of the symptoms of botulism in terms of a classic triad: bulbar palsy and descending paralysis, lack of fever, and clear senses and mental status ("clear sensorium").

After publishing on the neurological pathologies of aphasia, bulbar palsy, tabes dorsalis, and pathologies of the spinal cord, he achieved world fame with his 1885 work "On Emotions: A Psycho-Physiological Study".

Pseudobulbar palsy is a clinical syndrome similar to bulbar palsy but the damage is occurred in upper motor neurons, that is the nerves cells come down from the cerebral cortex inervating the motor nuclei in the medulla.

Progressive bulbar palsy patients that have this mutation are classified with FALS patients, Familial ALS (FALS) accounts for about 5%-10% of all ALS cases and is caused by genetic factors.

His greatest contributions were made in the myopathies that came to immortalize his name, Duchenne Muscular Dystrophy, Duchenne-Aran spinal muscular atrophy, Duchenne-Erb paralysis, Duchenne's disease (Tabes dorsalis), and Duchenne's paralysis (Progressive bulbar palsy).

In the United Kingdom, "motor neurone disease" (sometimes spelt as "motor neuron disease") refers both to amyotrophic lateral sclerosis and to the broader spectrum of related motor neuron disorders that includes progressive muscular atrophy, primary lateral sclerosis, and progressive bulbar palsy.

The leading proponent of Polish Stalinism from Kuźnica, Jan Kott, summarized the subject in the following way: "The Academy is like the monarchy; if it has existed for some time, one can get used to it, just as one can get accustomed to progressive bulbar palsy.

Bulbar palsy refers to impairment of function of the cranial nerves IX, X, XI and XII, which occurs due to a lower motor neuron lesion either at nuclear or fascicular level in the medulla oblongata or from lesions of the lower cranial nerves outside the brainstem.