basal membrane

Molecularly, the basal membrane is created by glycoprotein and proteoglycan protomers.

One such system has been developed to study skin development and basal membrane formation between keratinocytes and the stroma.

The basal membrane of these cells are characterized by tentacles like extensions that attach to astrocytes.

Intravasation refers to the invasion of cancer cells through the basal membrane into a blood or lymphatic vessel.

Moreover, the basal membrane component is expected to generate an additional physical and chemical barrier to axonal extensions.

Further, two independent networks are formed within the basal membrane by collagen IV and laminin for structural support.

On electron microscopy the mineral deposits appear as amorphous or crystalline material surrounded by a basal membrane.

Certain molecules, such as Integrins, localise specifically to the basal membrane and form connections with the extracellular matrix.

They are stored within the basal membrane or extracellular matric and released in an active form to stimulate tissue repair and healing.

The basal membrane is a histopathological extracellular matrix feature that forms at the center of injury and partially covers the astrocytic processes.

A numerable portion of infoldings occurs at the basal end of the dark cell toward the basal membrane.

Genes encoding for structural proteins of the basal membrane or extracellular matrix of the skeletal muscle fibres.

Other molecular components of the basal membrane include fibulin-1, fibronectin, entactin, and hepparan sulfate proteoglycan perlecan.

Ultimately, the astrocytes attach to the basal membrane, and the complex surrounds the blood vessels and nervous tissue to form the initial wound covering.

It also cleaves chondroitin sulfate, heparan sulfate and proteoglycans of the basal membrane.

In pancreatic development, netrin 1 is expressed in epithelial ductal cells and localizes to the basal membrane.

One is Goodpasture syndrome, an autoimmune disease whereby antibodies are directed against basal membrane antigens found in the kidney and lungs.

Invasion continues with the syncytiotrophoblasts reaching the basal membrane beneath the decidual cells, penetrating it and further invading into the uterine stroma.

Laminins of the basal membrane interact with the integrins of the growth cone to promote the forward movement of the growth cone.

The oocyst then develops under the basal membrane and after a period of two to three weeks a variable amount of sporozoites are produced within each oocyst.

Genes encoding for putative or demonstrated glycosyltransferases, that in turn affect the glycosylation of dystroglycan, an external membrane protein of the basal membrane.

Nearly all cases of CIS will continue to grow and progress until they begin to infiltrate and penetrate into and through the basal membrane or other/adjacent structures.

This compound crosses the basal membrane into the surrounding granulosa cells, where it is converted either immediately into estrone, or into testosterone and then estradiol in an additional step.

The population density of some of the anchoring fibers in basal membrane, collagen Ⅶ, for instance, is genetically determined, which points out that genetics may influence vocal fold health and pathogenesis.

Glucose is then transported across the apical membrane of the enterocytes by SLC5A1, and later across their basal membrane by SLC2A2.