atheromatous plaque

The atheromatous plaque is divided into three distinct components:

It is the precursor lesion of atheromas that may become atheromatous plaques.

D'Angelo et al(1978) found no prostacyclin generation in atheromatous plaques from three subjects.

Atherogenesis is the developmental process of atheromatous plaques.

In the context of heart or artery matters, atheromata are commonly referred to as atheromatous plaques.

The muscular portion of artery walls usually remain strong, even after they have remodeled to compensate for the atheromatous plaques.

In time, as cells die, this leads to extracellular calcium deposits between the muscular wall and outer portion of the atheromatous plaques.

An individual may develop a rupture of an atheromatous plaque at any stage of the spectrum of coronary artery disease.

The balloon is then inflated, and it compresses the atheromatous plaque and stretches the artery wall to expand.

Over a period of decades, chronically elevated serum cholesterol contributes to formation of atheromatous plaques in the arteries.

Laser endarterectomy : It is a technique in which an entire atheromatous plaque in the artery is excised.

It was Virchow in the middle of the nineteenth century who first put forward the concept of arterial wall injury leading to the development of the atheromatous plaque.

The release of mediators during allergic insults has been incriminated as a cause of coronary artery spasm and/or atheromatous plaque erosion or rupture.

Thus diminution of prostacyclin production and stimulation of platelet aggregation both mediated by lipid peroxides could contribute to thrombosis on atheromatous plaques.

This fork is a common site for atherosclerosis, an inflammatory buildup of atheromatous plaque that can narrow the lumen of the common or internal carotid arteries.

IVUS is used in the coronary arteries to determine the amount of atheromatous plaque built up at any particular point in the epicardial coronary artery.

The internal, common and external carotid arteries are clamped, the lumen of the internal carotid artery is opened, and the atheromatous plaque substance removed.

The main cause of heart attacks and the pain of angina is the lack of oxygen caused by blood clots and atheromatous plaque build up in the arteries.

Endarterectomy is a surgical procedure to remove the atheromatous plaque material, or blockage, in the lining of an artery constricted by the buildup of soft/hardening deposits.

Rupture of atheromatous plaque leads to occlusive thrombosis that produces myocardial ischaemia and cell death leading to loss of ventricular function and possibly death.

S. mutans is the most prevalent bacterial species detected in extirpated heart valve tissues as well as in atheromatous plaques, with an incidence of 68.6% and 74.1%, respectively.

After decades of progression, some of these atheromatous plaques may rupture and (along with the activation of the blood clotting system) start limiting blood flow to the heart muscle.

It is caused by an embolus lodged within the retinal vessel that originated from an atheromatous plaque in a more proximal (upstream) vessel, usually the internal carotid artery.

The ensuing inflammation leads to formation of atheromatous plaques in the arterial tunica intima, a region of the vessel wall located between the endothelium and the tunica media.

PON1 and PON3 prevent the formation of atherogenic oxidised-LDL, the form of LDL present in foam cells of an atheromatous plaque.