Apolipoprotein B-100 has been linked to the development of atherosclerosis.

One form is thought to be caused by mutated apolipoprotein B.

Similar results were observed in apolipoprotein A1 levels.

The protein encoded by this gene is a member of the apolipoprotein C family.

Other treatment options under investigation include infusion of recombinant human apolipoprotein A1.

Apolipoprotein tests are available in all hospital laboratories.

A gene that can carry a very high risk for the disease, the apolipoprotein E gene, was discovered in 1993.

It is associated with fibrinogen alpha chain, apolipoprotein A1, and lysozyme.

The drug accumulates in the liver, which is convenient since apolipoprotein B predominantly acts there.

Apolipoprotein(a) is the protein constituent of larger particles called lipoprotein(a).

The presence of the apolipoprotein E-4 gene, which increases the chances you will develop Alzheimer's disease.

Apolipoprotein B concentration falls substantially during treatment with lovastatin.

The genetic polymorphism of apolipoprotein E is associated with lipid abnormalities.

Apolipoprotein E polymorphism is associated with the progression of diabetic nephropathy.

In anti-cardiolipin-mediated autoimmune disease, there is a dependency on the apolipoprotein H for recognition.

These particles are then secreted into the lacteals in a process that depends heavily on apolipoprotein B-48.

Apolipoprotein synthesis in the intestine is regulated principally by the fat content of the diet.

Mice deficient in apolipoprotein B are more susceptible to invasive bacterial infection.

Apolipoprotein E polymorphism may influence the metabolism of lipoproteins in diabetic patients.

This protein - apolipoprotein (apo) E4 - is also the greatest known risk factor for Alzheimer's disease.

Almost all apolipoprotein, except apo B48 (produced by intestine)

APOO is a member of the apolipoprotein family.

Apolipoprotein E enhances proteolytic break-down of this peptide, both within and between cells.

An example of C-to-U editing is with the apolipoprotein B gene in humans.

Scientists of Genentech said their studies suggested that plasminogen and apolipoprotein(a) evolved from a common ancestral protein about 40 million years ago.