aortocaval

Move the patient onto her left side to reduce the risk of aortocaval compression.

Aortocaval compression is thought to be the cause of supine hypotensive syndrome.

Once the baby has been delivered aortocaval compression is no longer a problem, and further ephedrine is not usually required.

Four weeks of aortocaval shunt increased relative heart mass by 76% without a major change in body mass or systolic blood pressure.

Aortocaval fistula associated with ruptured aortic aneurysm.

The present study once again demonstrated that increased blood flow rate induced arterial enlargement in an aortocaval fistula model in rats.

The bleeding can be retroperitoneal or intraperitoneal, or the rupture can create an aortocaval or aortointestinal (between the aorta and intestine) fistula.

The electrical changes associated with myocardial hypertrophy induced by pressure overload (aortic constriction) were compared with those resulting from volume overload (aortocaval fistula).

This change appears 3 days after aortic constriction, 1 month after coronary artery ligation (in the healthy part of the left ventricle), and 1 month after an aortocaval fistula.

Aortocaval compression syndrome is compression of the abdominal aorta and inferior vena cava by the gravid uterus when a pregnant woman lies on her back, i.e. in the supine position.

Combined with a laparotomy, the procedure results in a Caesarean section that removes the aortocaval compression caused by the pregnant uterus, improving maternal chances of return of spontaneous circulation and potentially also delivering a viable neonate.

Typical sources of external pressure are an enlarged aorta (abdominal aortic aneurysm), the gravid uterus (aortocaval compression syndrome) and abdominal maligancies, such as colorectal cancer, renal cell carcinoma and ovarian cancer.

This prolongation affects the entire repolarization phase of the action potential 8 days after severe aortic constriction, after 8 days of isoproterenol treatment (5 mg/kg per day), and 3 months after an aortocaval fistula.

The present study determined whether changes in the activity and isoforms of protein kinase C (PKC) are associated with cardiac hypertrophy and heart failure owing to volume overload induced by aortocaval shunt (AVS) in rats.

The procedure should be considered within around four minutes after the onset of maternal cardiac arrest if basic and advanced life support attempts are proving unsuccessful at achieving return of spontaneous circulation, and her uterus is deemed capable of causing aortocaval compression.

A resuscitative hysterotomy, also known as a peri-mortem Caesarean delivery, is an emergency Caesarean delivery carried out where maternal cardiac arrest has occurred, to assist in resuscitation of the mother by removing the aortocaval compression generated by the gravid uterus.

The procedure should not be performed if the uterus is not judged to be big enough to cause maternal haemodynamic changes through aortocaval compression, as there is no potential benefit to the mother and the fetus or fetuses will not be viable in such an early stage of pregnancy.