angioid streak

Angioid streaks themselves do not cause distortion of vision, even if they cross into the foveal area.

Associated conditions such as angioid streaks and retinitis pigmentosa should be screened for.

In 1889, he was the first physician to describe angioid streaks, a disorder that affects Bruch's membrane.

Angioid streaks may develop, possibly as a result of calcification of collagen or other pathological deposition.

Eventually the mineralization of the elastic fibers in the Bruch membrane create cracks (angioid streaks) that radiate out from the optic nerve.

The diagnostic criteria for PXE are the typical skin biopsy appearance and the presence of angioid streaks in the retina.

A few years later, ophthalmologist Hermann Jakob Knapp called them "angioid streaks" because of their resemblance to blood vessels.

The inherited disorder pseudoxanthoma elasticum may lead to angioid streaks with calcification of Bruch's membrane, the elastic tissue below the retinal ring.

His name is lent to the eponymous "Knapp streaks"; also known as angioid streaks, which are tiny breaks in the elastin-filled tissue in the back of the eye.

Certain conditions have been associated with disc drusen such as retinitis pigmentosa, angioid streaks, Usher syndrome, Noonan syndrome and Alagille syndrome.

Angioid streaks, also called Knapp streaks or Knapp striae are small breaks in Bruch's membrane, an elastic tissue containing membrane of the retina that may become calcified and crack.

Angioid streaks are often associated with pseudoxanthoma elasticum (PXE), but have been found to occur in conjunction with other disorders, including Paget's disease, sickle cell anemia and Ehlers-Danlos Syndrome.

Examples include the skin of the breast in inflammatory breast cancer, or breaks in Bruch's Membrane called angioid streaks, which are common in pseudoxanthoma elasticum, or in elephantiasis caused by thread-like, microscopic parasitic worms (filariasis).