alveolar macrophage

The other 10% is taken up by alveolar macrophages and digested.

Alveolar macrophages interact with P. carinii in the lung as the first line of defense against infection.

Under normal circumstances, alveolar macrophages and other lung cells produce a variety of chemokines.

Activity of the alveolar macrophage is relatively high, because they are located at one of the major boundaries between the body and the outside world.

This crosses the bone marrow barrier to the blood and is consumed in the lung by alveolar macrophages.

When in the lungs, M. tuberculosis is taken up by alveolar macrophages, but they are unable to digest the bacterium.

Upon inhalation the bacteria can infect alveolar macrophages.

Alveolar macrophages phagocytize and destroy conidia within their phagosomes.

Inhaled virulent strains of R. equi are phagocytosed by alveolar macrophages.

The encoded protein can function as an elastase over a broad pH range in alveolar macrophages.

The interaction of alveolar macrophages with P. carinii is mediated by fibronectin [ 6].

Alveolar macrophages are phagocytes that play a critical role in homeostasis, host defense, the response to foreign substances, and tissue remodeling.

Two positions are available in the Department of Respiratory Medicine to study gene activation in human alveolar macrophages.

Thymomodulin increases the depressed production of superoxide anion by alveolar macrophages in patients with chronic bronchitis.

Alveolar macrophages are also involved in the phagocytosis of apoptotic and necrotic cells that have undergone cell-death.

Alveolar macrophages are frequently seen to contain granules of exogenous material such as particulate carbon that they have picked up from respiratory surfaces.

Alveolar macrophages release numerous secretory products and interact with other cells and molecules through the expression of several surface receptors.

Alveolar macrophages (dust cells) ingest the red blood cells, and become engorged with brownish hemosiderin.

It found that when alveolar macrophages were exposed to unfiltered smoke, their ability to fight bacteria was reduced, unlike exposure to water-filtered smoke.

Haptoglobin-alpha is highly expressed in alveolar macrophages and eosinophils in inflamed human lung tissues [ 33].

Specific macrophage subpopulations are associated with different tissues (alveolar macrophages in the lung, microglial cells in the central nervous system, etc.).

TB infection begins when the mycobacteria reach the pulmonary alveoli, where they invade and replicate within endosomes of alveolar macrophages.

An alveolar macrophage (or dust cell) is a type of macrophage found in the pulmonary alveolus, near the pneumocytes, but separated from the wall.

Since alveolar macrophages are pivotal regulators of local immunological homeostasis, their population density is decisive for the many processes of immunity in the lungs.

In normal conditions, alveolar macrophages adhere closely to alveolar epithelial cells, thus inducing the expression of the αvβ6 integrin.