The list of compounds that can be used to treat allodynia is even longer than this.

Some are specific for certain types of allodynia while others are general.

Since 1979, this stimulus induced pain is to be called allodynia in medicine.

Different cell types have also been linked to allodynia.

Most patients experiencing allodynia, experience pain with touch and pressure, however some can be hypersensitive to temperature.

Temperature or physical stimuli can provoke allodynia, and it often occurs after injury to a site.

This mechanism also explains the perpetuation of sensitization and thus allodynia.

The presence of mechanical allodynia, hinders other physical treatments.

Subcutaneous sumatriptan as a successful treatment for migraine associated with allodynia.

Dose response of intrathecal adenosine in experimental pain and allodynia.

The cell types involved in nociception and mechanical sensation are the cells responsible for allodynia.

This should not be confused with allodynia, where normally non-painful stimuli evoke pain.

These results indicate that the rats exhibited hyperalgesia, allodynia, and dysesthesia.

There are different kinds or types of allodynia:

States of neuropathic pain, such as hyperalgesia and allodynia, are also directly related to mechanosensation.

It has analgesic effects in animal studies, particularly against "atypical" pain such as hyperalgesia and allodynia.

Burning and tingling can also be accompanied by hypersensitivity, usually in the form of dysaesthesia or allodynia.

Severe cases of ciguatera can also result in cold allodynia, which is a burning sensation on contact with cold.

The changes in this note are the specification of allodynia and the inclusion of hyperalgesia explicitly.

Hyperalgesia and allodynia are examples of neuropathic pain.

It is believed that restructured synaptic connections in the spinal cord are responsible for allodynia.

Both of these mechanisms for pain modulation have been implicated in the pathology of allodynia.

It can also help treat migraines in the setting of allodynia, where it is more effective than triptans.

It also increases allodynia by interfering with the action of the κ-opioid peptide dynorphin.

Pain associated with allodynia can be attributed to myelinated A-fibers as a result of a change in their central functional connectivity.