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As an example, one study involving rats found that having narrowed afferent arterioles contributed to the development of increased blood pressure.
This occurs near the site of contact between the thick ascending limb and the afferent arteriole.
The afferent arterioles later diverge into the capillaries of the glomerulus.
The afferent arterioles, then, enter Bowman's capsule and end in the glomerulus.
The afferent arterioles are a group of blood vessels that supply the nephrons in many excretory systems.
A glomerulus receives its blood supply from an afferent arteriole of the renal circulation.
If the afferent arterioles are constricted then the blood pressure in the capillaries of the kidneys will drop.
In their outward course, they give off lateral branches, which are the afferent arterioles that supply the renal corpuscles.
Prostaglandins normally cause vasodilation of the afferent arterioles of the glomeruli.
The vascular pole is the side with the afferent arteriole and efferent arteriole.
The walls of the afferent arteriole contain specialized smooth muscle cells that synthesize renin.
The afferent arterioles form the glomerulus (network of capillaries enclosed in Bowman's capsule).
Prostaglandins and NO also vasodilate the afferent arterioles.
These cells are similar to epithelium and are located in the media of the afferent arterioles as they enter the glomeruli.
Further, the macula densa releases adenosine which causes constriction of the afferent arterioles.
Adenosine is responsible for constriction of afferent arteriole and reduction in GFR.
Histologically, glomus tumors are made up of an afferent arteriole, anastomotic vessel, and collecting venule.
Blood flows into these capillaries through the afferent arteriole and leaves through the efferent arteriole.
At the vascular pole, the afferent arterioles and efferent arterioles enter the Bowman's capsule.
Sympathetic nervous system action as well as hormones can also impact glomerular filtration rate by modulating afferent arteriole diameter.
Additionally, vasopressin selectively contracts efferent arterioles probably through the VR, but not the afferent arteriole.
When there is an increase in the salt concentration, the cells secretes a locally-active vasopressor, which acts on the adjacent afferent arteriole to cause constriction.
Each arcuate artery supplies several interlobular arteries that feed into the afferent arterioles that supply the glomerulus.
The point where DCT contacts afferent arteriole of renal corpuscle is called macula densa.
Constriction of the afferent arterioles causes a decrease in the intraglomerular pressure, reducing GFR proportionally.