adrenal androgen

Adrenal androgens contribute to the androgenic events of early puberty in girls.

Effects of adrenal androgens on the transplantable human prostate tumor PC-82.

Puberty is preceded by adrenarche, marking an increase of adrenal androgen production between ages 6-10.

Dehydroepiandrosterone, an adrenal androgen, increases human foam cell formation: a potentially pro-atherogenic effect.

Ovarian and adrenal androgen production is minimal and produces little pubic or other body hair.

Women in or approaching menopause may develop acne because their adrenal androgens are no longer adequately countered by female hormones.

Researchers have unsuccessfully tried to identify a new pituitary peptide, to be called "adrenal androgen stimulating hormone".

Similarly, DHT drops, while adrenal androgen and cortisol will increase with the stress response.

In other words, glucocorticoid replacement is the primary method of reducing the excessive adrenal androgen production in both sexes.

Adrenal androgen and gonadal androgen result in pubic and axillary hair.

Glucocorticoids can be provided at minimal replacement doses because there is no need for suppression of excessive adrenal androgens or mineralocorticoids.

Adrenal androgens contribute to the development of pubic hair (pubarche), adult body odor, and other androgenic changes in both sexes.

The primary goals of hormone replacement are to protect from adrenal insufficiency and to suppress the excessive adrenal androgen production.

Tamoxifen blocks the estrogen receptor so the remaining estrogen you make (from peripheral conversion of adrenal androgens) doesn't have any effect.

On the other hand, adrenal androgens are readily converted to estradiol, which accelerates bone maturation and can lead to early epiphyseal closure.

Adrenal androgens are steroids produced by the zona reticulosa of the adrenal cortex in both sexes.

Medications that block the production of adrenal androgens such as DHEA include ketoconazole and aminoglutethimide.

Pubic hair can develop from adrenal androgens alone, and can develop even when the ovaries or testes are defective and nonfunctional.

In girls, the adrenal androgens of adrenarche produce most of the early androgenic changes of puberty: pubic hair, body odor, skin oiliness, and acne.

ACTH partially regulates adrenal androgen secretion, also CRH.

Some androgens are called adrenal androgens because they are made in the adrenal cortexes of the adrenal glands.

Oral dehydroepiandrosterone for adrenal androgen replacement: pharmacokinetics and peripheral conversion to androgens and estrogens in young healthy females after dexamethasone suppression.

DHT has two to three times greater androgen receptor affinity than testosterone and has 15-30 times greater affinity than adrenal androgens.

In congenital adrenal hyperplasia, the adrenal cortex lacks appropriate corticosteroid synthesis, leading to excess adrenal androgens and affects female fetuses.