Sneddon's syndrome

The treatment of Sneddon's syndrome includes daily aspirin and low dose heparin, which is a blood thinner.

Sneddon's syndrome is a rare condition that occurs in families and may be inherited in an autosomal dominant fashion.

Sneddon's Syndrome most often becomes apparent in women in their thirties, though cases do occur in men and in children.

Sneddon's syndrome manifests with elevated blood pressure (hypertension), a skin rash (livedo reticularis), and cerebrovascular disease.

Sneddon's syndrome may instead present with livedo racemosca, which involves larger, less organized patches of bluish-purple mottling of the skin.

Both are generally found first in the extremities, both worsen in cold and either may occur without Sneddon's Syndrome or any other systemic disease.

Sneddon's syndrome (also known as "Idiopathic livedo reticularis with cerebrovascular accidents") is a form of arteriopathy characterized by several symptoms, including:

Sneddon's Syndrome was formerly understood to be a type of autoimmune disease called antiphospholipid syndrome, although it has been reclassified as a noninflammatory cerebrovascular disease.

Livedo Reticularis and Cerebrovascular Accidents Sneddon's Syndrome None Sneddon syndrome is a rare progressive disorder affecting the blood vessels characterized by the association of a skin condition and neurological abnormalities.

Currently, Sneddon's Syndrome is understood as a progressive, noninflammatory arteriopathy leading to the characteristic skin condition and to cerebrovascular problems including stroke, transient ischemic attack (TIA), severe but transient neurological symptoms thought to be caused by cerebral vasospasm, and early onset dementia.