Purkinje fibers

The effect upon Purkinje fibers and ventricles is negligible.

Purkinje fibers also have the ability of firing at a rate of 15-40 beats per minute if left to their own devices.

These bundles and fascicles give rise to thin filaments known as Purkinje fibers.

U waves are thought to represent repolarization of the papillary muscles or Purkinje fibers.

Purkinje fibers are a unique cardiac end-organ.

The electrical origin of atrial Purkinje fibers arrives from the sinoatrial node.

There is a near total loss of Purkinje fibers in the cerebellar hemisphere and an undetectable dentate nucleus.

The two bundle branches taper out to produce numerous Purkinje fibers, which stimulate individual groups of myocardial cells to contract.

This current can be seen in Phase 2 of action potential of atrial, ventricular and Purkinje fibers.

While in the ventricles, the signal is carried by specialized tissue called the Purkinje fibers which then transmit the electric charge to the myocardium.

Purkinje fibers take up stain differently from the surrounding muscle cells, and, on a slide, they often appear lighter and larger than their neighbors.

Specialized muscle fibers called Purkinje fibers then conduct the signals to the apex of the heart along and throughout the ventricular walls.

Purkinje fibers allow the heart's conduction system to create synchronized contractions of its ventricles, and are therefore essential for maintaining a consistent heart rhythm.

Automaticity is also increased, in the atria, AV node, Purkinje fibers and ventricles.

From the branches, the impulse travels through the Purkinje fibers and allows the electrical impulse to end in the ventricles to initiate ventricular depolarization.

The Purkinje fibers (Purkyne tissue or subendocardial branches) are located in the inner ventricular walls of the heart, just beneath the endocardium.

Given no aberrant channels, the atrial and ventricular Purkinje fibers are distinctly shielded from each other by collagen or the cardiac skeleton.

Action potentials from the AV node travel through the bundle of His and thence to the Purkinje fibers.

Current then passes from the atria through the bundle of His, from which it travels along Purkinje fibers to reach and depolarize the ventricles.

The Purkinje fibers are further specialized to rapidly conduct impulses (numerous fast voltage-gated sodium channels and mitochondria, fewer myofibrils than the surrounding muscle tissue).

It was demonstrated in this study that the basket cells which encapsulate some regions of Purkinje fibers can cause inhibitory effects on the Purkinje cells.

Unlike most antiarrhythmics, BRL-32872's effects are homogeneous within the various cardiac tissue types (nodal cells, cardiomyocytes, Purkinje fibers).

If the AV node also fails, Purkinje fibers (or known by some as the bundle of His) are capable of acting as the pacemaker.

In normal conduction, the impulse would travel across the "bundle of His" (AV bundle), down the bundle branches, and into the Purkinje fibers.

They include: hypotonia, developmental delay, mental retardation, degeneration of axons, sensory neuropathy, tremors, demyelinization, gray matter anomalies, myoclonic seizures, and loss of purkinje fibers.