Purkinje cell

Each climbing fiber will form synapses with 1-10 Purkinje cells.

Others are studies of neurons, such as purkinje cells.

"Purkinje cells are very big, and they sort of hit you in the eye in the microscope," he said.

This area produces Purkinje cells and deep cerebellar nuclear neurons.

They terminate directly on Purkinje cells as the climbing fiber input system.

Purkinje cells are aligned like dominos stacked one in front of the other.

Chronic ethanol consumption results in a loss of Purkinje cell synapses.

A defining characteristic of Purkinje cells is the apical dendrite.

Put simply, without Purkinje cells, an animal loses its sense of space and distance, making balance and coordination difficult.

Discovery of the P-type calcium channel in the Purkinje cells.

The sodium-potassium pump has been shown to control and set the intrinsic activity mode of Purkinje cells.

Climbing fibers project to Purkinje cells and also send collaterals directly to the deep nuclei.

A microzone is defined as a group of Purkinje cells all having the same somatotopic receptive field.

The maxi-channels targeted have their highest density in the cerebellar Purkinje cells.

Purkinje cells are found within the Purkinje layer in the cerebellum.

On the converse, a single deep nuclear cell receives input from approximately 860 Purkinje cells (again in cats).

The Purkinje cells send their afferents to the ipsilateral dentate nucleus.

It develops when the neurons known as Purkinje cells, located in the cerebellum of the brain, begin to die off.

Purkinje cells show two distinct forms of electrophysiological activity:

Purkinje cells are especially susceptible to ethanol excitotoxicity.

The most notable exception is the direct inhibition of the vestibular nuclei by Purkinje cells.

Ataxia, the most common symptom, is due to misfiring of Purkinje cells in the cerebellum.

Plasticity of the synapse between a parallel fiber and a Purkinje cell is believed to be important for motor learning.

High calcium concentrations in the post-synaptic Purkinje cells is a necessity for the induction of long-term depression.

Purkinje cells normally emit action potentials at a high rate even in the absence of synaptic input.