It is also an active component of programmed cell death.

Programmed cell death is also necessary to start the process of menstruation.

Apoptosis (programmed cell death) appears to play a large role.

This result casts doubt on the growth control and programmed cell death hypotheses.

It is also essential for development of nerval system by programmed cell death.

Programmed cell death (apoptosis) is a normal part of species development.

Biologists who study the development of the embryo were the first to describe programmed cell death.

Their observations helped to lead later work toward the genetic pathways of programmed cell death.

Cells with reduced Pdx1 have an increased rate of apoptotic programmed cell death.

The gas can also induce a type of programmed cell death known as apoptosis.

This will cause the tumor to undergo apoptosis, which is programmed cell death.

Programmed cell death had been known long before "the worm people" began to publish their celebrated findings.

These changes reflect delayed apoptosis or a failure of programmed cell death.

When programmed cell death does not work properly, cells that should be eliminated can become immortal.

He said better understanding of programmed cell death might lead to development of treatments for cancer and other diseases.

These B cells are believed to undergo programmed cell death or apoptosis.

These effects block apoptosis, or normal programmed cell death.

Apoptosis is a form of programmed cell death in multicellular organisms.

Many cells undergo programmed cell death, or apoptosis, during fetal development.

Also known as programmed cell death and cell suicide.

For example, depolarization of the plasma membrane appears to be an important step in programmed cell death.

Apoptosis, or programmed cell death, is largely influenced by the mitochondria.

Programmed cell death - similar to the above function, although individuals must have variable stress survival level to prevent entire population destruction.

In programmed cell death interphase is followed or preempted by apoptosis.

Programmed cell death removes the "webbing" between the digits and joints.

Mutations in the C14orf104 are associated with primary ciliary dyskinesia.

Rhinorrhea can also be the side effect of several genetic disorders, such as primary ciliary dyskinesia.

Primary ciliary dyskinesia occurs in approximately 1 in 25 of Totalis.

Certain genetic conditions can also cause bronchiectasis, including primary ciliary dyskinesia and immunodeficiency syndromes.

Primary ciliary dyskinesia.

Low levels have been found in primary ciliary dyskinesia, bronchopulmonary dysplasia, and pulmonary arterial hypertension.

Primary ciliary dyskinesia, which affects the mobility of cilia in the lungs, aids in the development of the disease.

About 25% of individuals with situs inversus have an underlying condition known as primary ciliary dyskinesia (PCD).

Some common ciliopathies include primary ciliary dyskinesia, hydrocephalus, polycystic liver and kidney disease, and some forms of retinal degeneration.

Two other types, primary ciliary dyskinesia, and biliary dyskinesia, are caused by specific kinds of ineffective movement of the body, and are not movement disorders.

Genetic mutations compromising the proper functioning of cilia, ciliopathies, can cause chronic disorders such as primary ciliary dyskinesia (PCD), nephronophthisis or Senior-Loken syndrome.

Of interest, there is an association of primary ciliary dyskinesia with left-right anatomic abnormalities such as situs inversus (a combination of findings known as Kartagener's syndrome) and other heterotaxic defects.

Mutations in this gene have been implicated in causing primary ciliary dyskinesia (PCD, formerly called 'immotile cilia syndrome') and Kartagener syndrome (PCD with situs inversus totalis).

Currently recognized ciliopathies include Joubert syndrome, primary ciliary dyskinesia (also known as Kartagener Syndrome), Bardet-Biedl syndrome, polycystic kidney disease and polycystic liver disease, nephronophthisis, Alstrom syndrome, Meckel-Gruber syndrome and some forms of retinal degeneration.

It can be caused by chronic meningitis and neurosyphilis that would increase intracranial pressure over a long period of time, and in some cases by ciliopathy including ciliopathy due to primary ciliary dyskinesia (Kartagener syndrome, Afzelius' syndrome or Siewert's syndrome).

Diseases that prevent the cilia from working properly, such as Kartagener syndrome and immotile cilia syndrome.

Its use has been largely supplanted by immunohistochemistry, but it is still in common use for certain tasks, including the diagnosis of kidney disease and the identification of immotile cilia syndrome among many others.

Mutations in this gene have been implicated in causing primary ciliary dyskinesia (PCD, formerly called 'immotile cilia syndrome') and Kartagener syndrome (PCD with situs inversus totalis).