Idiopathic Basal Ganglia Calcification, also known as Fahr disease or Fahr's Syndrome is a rare, genetically dominant, inherited neurological disorder characterized by abnormal deposits of calcium in areas of the brain that control movement.

Fahr disease, formerly FIBGC Cerebrovascular Ferrocalcinosis striopallidodentate calcinosis None Familial idiopathic basal ganglia calcification (FIBGC) is a rare neurological disorder characterized by the presence of abnormal calcium deposits (calcifications) of unknown cause.

There is currently no cure for Fahr's Syndrome, nor a standard course of treatment.

The prognosis for any individual with Fahr's Syndrome is variable and hard to predict.

Mutations in the SLC20a2 gene are associated with idiopathic basal ganglia calcification (Fahr's syndrome).

Such symptoms are often present in those who suffer from dystonia, athymhormic syndrome, Fahr's syndrome, Huntington's disease or Parkinson's disease.

Idiopathic Basal Ganglia Calcification, also known as Fahr disease or Fahr's Syndrome is a rare, genetically dominant, inherited neurological disorder characterized by abnormal deposits of calcium in areas of the brain that control movement.

Idiopathic Basal Ganglia Calcification, also known as Fahr disease or Fahr's Syndrome is a rare, genetically dominant, inherited neurological disorder characterized by abnormal deposits of calcium in areas of the brain that control movement.

Fahr disease, formerly FIBGC Cerebrovascular Ferrocalcinosis striopallidodentate calcinosis None Familial idiopathic basal ganglia calcification (FIBGC) is a rare neurological disorder characterized by the presence of abnormal calcium deposits (calcifications) of unknown cause.

Mutations in the SLC20a2 gene are associated with idiopathic basal ganglia calcification (Fahr's syndrome).

Important It is possible that the main title of the report Familial Idiopathic Basal Ganglia Calcification is not the name you expected.

This association suggests that familial idiopathic basal ganglia calcification is caused by changes in phosphate homeostasis, since this gene encodes for PIT-2, an inorganic phosphate transporter.

Idiopathic Basal Ganglia Calcification, also known as Fahr disease or Fahr's Syndrome is a rare, genetically dominant, inherited neurological disorder characterized by abnormal deposits of calcium in areas of the brain that control movement.

Fahr disease, formerly FIBGC Cerebrovascular Ferrocalcinosis striopallidodentate calcinosis None Familial idiopathic basal ganglia calcification (FIBGC) is a rare neurological disorder characterized by the presence of abnormal calcium deposits (calcifications) of unknown cause.

There is currently no cure for Fahr's Syndrome, nor a standard course of treatment.

The prognosis for any individual with Fahr's Syndrome is variable and hard to predict.

Mutations in the SLC20a2 gene are associated with idiopathic basal ganglia calcification (Fahr's syndrome).

Such symptoms are often present in those who suffer from dystonia, athymhormic syndrome, Fahr's syndrome, Huntington's disease or Parkinson's disease.

Idiopathic Basal Ganglia Calcification, also known as Fahr disease or Fahr's Syndrome is a rare, genetically dominant, inherited neurological disorder characterized by abnormal deposits of calcium in areas of the brain that control movement.