Anti-Müllerian hormone

In most males, the development of the embryo into a female is prevented by Anti-Müllerian hormones.

Aromatase activity is decreased by prolactin, anti-Müllerian hormone.

Other hormonal changes in chemotherapy include decrease in inhibin B and anti-Müllerian hormone levels.

Müllerian inhibiting hormone, an alternative name for Anti-Müllerian hormone.

Anti-Müllerian hormone causes the Müllerian ducts to regress.

They secrete anti-Müllerian hormone, testosterone, and Dihydrotestosterone.

This is due to the production of Anti-Müllerian hormone by the Sertoli cells of the testes.

Another significant hormone in sexual differentiation is the Anti-müllerian hormone, which inhibits development of the Müllerian ducts.

Externally, the animal appears female, but various aspects of female reproductive development are altered due to acquisition of anti-Müllerian hormone from the male twin.

The testes secrete anti-Müllerian hormone around this time to suppress the development of the Müllerian ducts, and cause their degeneration.

Circulating Anti-Müllerian hormone (AMH) can predict excessive and poor response to ovarian stimulation.

Reference ranges for Anti-Müllerian hormone, as estimated from reference groups in the United states, are as follows:

The first phase, movement across the abdomen to the entrance of the inguinal canal appears controlled (or at least greatly influenced) by anti-müllerian hormone (AMH).

Anti-Müllerian hormone receptor is a receptor for anti-Müllerian hormone.

Without this anti-Müllerian hormone, the Müllerian ducts develop into the female internal genitalia (uterus, cervix, fallopian tubes, and upper vaginal barrel).

It acts during chondrocyte differentiation and, with steroidogenic factor 1, regulates transcription of the anti-Müllerian hormone (AMH) gene.

However, it appears that repetitive oocyte donation cycles does not cause accelerated ovarian aging, evidenced by absence of decreased anti-Müllerian hormone (AMH) in such women.

Other hormones produced by the gonads which may be decreased by hypogonadism include progesterone, DHEA, anti-Müllerian hormone, activin, and inhibin.

Anti-Müllerian hormone also known as AMH is a protein that, in humans, is encoded by the AMH gene.

Testicular function can also be assessed by measuring serum anti-Müllerian hormone levels, which in turn can further differentiate PAIS from gonadal dysgenesis and bilateral anorchia.

This family of cytokines and hormones include activin, Anti-müllerian hormone (AMH), bone morphogenetic proteins (BMPs), and Nodal.

Activin, inhibin and a number of other structurally related proteins such as anti-Müllerian hormone, bone morphogenetic protein, and growth differentiation factor belong to the TGF-β protein superfamily.

The anti-Müllerian hormone receptor (Müllerian Inhibiting Substance Type II Receptor) can be responsible for persistent Müllerian duct syndrome.

The testes begin to secrete three hormones that influence the male internal and external genitalia: they secrete anti-Müllerian hormone (AMH), testosterone, and dihydrotestosterone (DHT).