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There are two cases of acute toxicity known in man.
Unfortunately, use of acute toxicity tests remains widespread in industry.
There are several different types of acute toxicity tests.
The acute toxicity of the low calcium Locke's solution was also investigated.
These pose a greater threat of acute toxicity to animals (and people) especially in the first 24 hours after application.
The acute toxicity of the drug is low.
To be known as acute toxicity, the bad effects should happen within 14 days of the substance thouching something.
Sulpiride has a relatively low order of acute toxicity.
Its acute toxicity is comparable to some nerve agents.
The results from acute toxicity tests can thus help determine whether the effect identified is due to a specific contaminant.
At high enough doses, however, acute toxicity and death can occur through the same mechanism as other nerve agents.
Dicamba was tested for acute toxicity in a variety of aquatic animals.
Manganese may affect liver function, but the threshold of acute toxicity is very high.
It has low to moderate acute toxicity, with kidney and liver effects as the main hazard concerns.
Traces of about twenty different drugs were found, along with acute toxicity (perhaps caused by a stimulant).
It can even cause acute toxicity in humans if exposed for long periods of time or a sufficient dose.
There is very little known about acute toxicity for humans, but there have been animal studies, showing the following results.
Of course, this is the acute toxicity.
The acute toxicity of chlorfenvinphos varies widely between species.
Triphenylphosphate exhibits low acute toxicity by dermal or oral contact.
In some regulatory systems these acute toxicity categories may be subdivided or extended for certain sectors.
It has moderate acute toxicity by the oral and inhalation routes in rats.
Now we are removing acute toxicity category five. Why are we doing this?
Like the phallotoxins they do not exert any acute toxicity after ingestion in humans.
In normal conditions, humans have the ability to metabolize glutamate that has a very low acute toxicity.