Some patients rapidly recover from acute lung injury, and have no permanent sequelae.

Neutrophils also play a key role in the development of most forms of acute lung injury.

It can result from acute lung injury or a lung disease like emphysema.

A less severe form is called acute lung injury (ALI).

It can occur without a known cause or as the result of a lung disease or acute lung injury.

Severe exposure may result in acute lung injury which may not be present until several hours after exposure.

Elevated NO2 may cause acute lung injury.

Mixtures of other cleaning agents and or organic matter can result in a gaseous reaction that can cause acute lung injury.

Specifically in sheep, intravenous administration of oleic acid causes acute lung injury with corresponding pulmonary edema.

The physiologic shunt grows much larger in acute lung injury and especially in adult respiratory distress syndrome.

Primary influenza pneumonia occurs most commonly in adults and may progress rapidly to acute lung injury requiring mechanical ventilation.

The presence of this virus in acute lung injury and exacerbations of idiopathic lung fibrosis has been reported.

Acute lung injury (including ARDS, trauma)

Transfusion-associated acute lung injury (TRALI) is an increasingly recognized adverse event associated with blood transfusion.

In one study of patients recovering from acute lung injury in intensive care, those patients who developed hypoglycemia while hospitalized showed an increased rate of depression.

In the case of non-allergic acute lung injury in the setting of metal fume fever, a standard or even recommended approach to treatment has not been studied.

Lung diseases, like chronic obstructive pulmonary disease (COPD), tuberculosis, and acute lung injury, cause pneumothorax.

ARDS may also be called acute lung injury, non-cardiac pulmonary edema, and increased-permeability pulmonary edema.

The production of PF24 began in response to an increase in reported cases of transfusion-related acute lung injury, or TRALI.

This is a historical term for acute lung injury (ALI) or acute respiratory distress syndrome (ARDS).

In patients with acute lung injury and acute respiratory distress syndrome conventional humidifiers are preferred to HMEs for improved elimination of carbon dioxide.

Acute lung injury and the more severe acute respiratory distress syndrome (ARDS) is marked by hypoxemia due to impaired ventilation-perfusion matching.

In animal studies, NO dose-response curves depend on the model of acute lung injury and on the pathophysiology of pulmonary artery hypertension [ 33, 34, 35].

Transfusion-related acute lung injury (TRALI) is an uncommon syndrome that is due to the presence of leukocyte antibodies in transfused plasma.

HO-1 is a Nrf2 target gene that has been shown to protect from a variety of pathologies, including sepsis, hypertension, atherosclerosis, acute lung injury, kidney injury, and pain.

Acute limb ischaemia can occur in patients through all age groups.

Acute limb ischaemia is due to either an embolism or thrombosis.

In treating acute limb ischaemia time is everything.

Patients that smoke and have diabetes mellitus are at a higher risk of developing acute limb ischaemia.

Acute limb ischaemia occurs when there is a sudden lack of blood flow to a limb.

Symptoms of acute limb ischaemia include:

The major cause of acute limb ischaemia is arterial thrombosis (85%), while embolic occlusion makes up 15% of causes.

In addition to critical limb ischaemia, and acute limb ischaemia, there are several other related ischemic conditions which exist.

With proper surgical care, acute limb ischaemia is a highly treatable condition; however, prolonged or delayed treatment can result in morbidity, amputation, and/or death.

Arterial and venous occlusions, including carotid artery stenosis, pulmonary embolisms and acute limb ischaemia.

In the worst cases acute limb ischaemia progresses to critical limb ischaemia, and results in death or limb loss.

Compartment syndrome can occur because of acute limb ischaemia because of the biotoxins that accumulate distal to the occlusion resulting in edema.

In order to treat acute limb ischaemia there are a series of things that can be done to determine where the occlusion is located, the severity, and what the cause was.